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The anti-RA mechanism of ginsenoside Rg3 is related to its efficacy in regulating the oxidative phosphorylation pathway, and enhancing the ability of CD4 + CD25 + Foxp3 + Treg cells to maintain peripheral immune tolerance, thereby resisting inflammation and immunosuppression (132)

It belongs to a novel class of agents known as triple agonists, meaning it simultaneously activates three hormone receptors: glucagon-like peptide-1 (GLP-1), glucose-dependent insulinotropic polypeptide (GIP), and glucagon receptors

Importantly, Akt/PKB also phosphorylates forkhead transcription factors of the FOXO subfamily, and this leads to their inactivation and retention in cytoplasm[254, 255] resulting in a reduced transcriptional activity[256]