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In case of diabetes, high blood glucose and oxidative stress interfere with platelet aggregation and clotting factors due to hyperglycemia and advanced glycation products, which increase the sensitivity of platelets for spontaneous aggregation, causing rapid consumption and increased probability of reduced availability during hemostasis (Rodriguez and Johnson, 2020) and cause endothelial dysfunction, interfering with hemostasis (Patel et al., 2019)

While the effects of GIP receptor antagonism on these outcomes remain unknow, GIPR knockout mice have been shown to exhibit impaired learning and memory, as well as reduced synaptic plasticity (111)

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Thus, NADPH negatively correlated with sedoheptulose 1,7-bisphosphate (SBP, r = 0.51