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Methotrexate-induced liver injury is associated with oxidative stress, impaired mitochondrial respiration, and endoplasmic reticulum stress in vitro

Since the arrestins can also mediate their own G protein-independent signaling, any given GPCR can theoretically signal (predominantly) either via G proteins or arrestins, i.e., be a G protein- or arrestin-biased receptor, depending on the bound ligand

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