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The results demonstrated that BPC 157 caused a significant reduction in all of the necrosis markers commonly measured in routine practice (i.e., CK, CK-MB, LDH, and cTnT) and attenuated gross (no visible infarcted area) and histological damage, including ECG (no ST-T ischemic changes) and echocardiography (preservation of systolic left ventricular function) damage induced by isoprenaline

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Antioxidant Signaling and ROS Attenuation Studies in aged rodent models demonstrated that Epitalon treatment reduced markers of oxidative stress, including lipid peroxidation products (MDA) and protein carbonylation, while upregulating superoxide dismutase (SOD) and catalase activity