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Certain nerve damage may never be fully repaired, especially if youve gone more than six months without treatment

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Since glutathione deficiency and oxidative stress often contribute to nervous system conditions, glutathione supplementation may reduce symptoms and slow disease progression

Hypothetical Molecular Mechanisms Underlying Hyperhomocysteinemia-Mediated Vascular Disorders Hyperhomocysteinemia-Mediated Oxidant Stress through Imbalanced Thiol Redox Status Numerous studies indicate that oxidant stress plays a role in the development of occlusive atherosclerotic lesions that are observed primarily in homozygotes for the CBS defect (McCully, 1983) and later in experimental hyperhomocysteinemia