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by contrast, U0126, an inhibitor of MAPK kinases, suppressed PFOS-mediated IRE1 overexpression, suggesting that ER stress is a downstream target of the ERK pathway (Fig

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VKORC1L1 overexpression significantly attenuated tumor growth suppression in p53 wild-type tumors but not in p53-null tumors, suggesting a new pathway for p53-controlled ferroptosis and tumor growth inhibition mediated by vitamin K metabolism modulation