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cataloged 36 BPC-157 studies from 1993 to 2025, confirming that tendon and ligament outcomes are among the most reproducible findings in the BPC-157 literature
They found that BPC-157 enhanced the outgrowth of tendon fibroblasts from explants, increased their survival under oxidative stress (HO challenge), and promoted FAK-paxillin phosphorylation, collectively suggesting a direct effect on the key cell type involved in tendon repair
Hydration and gradual dose escalation may minimize this concern
The information provided reflects current available evidence and should not be used to self-diagnose or self-treat
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