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IBD is a multifactorial disease where activation of inflammatory cells are thought to play a key role for its development, however the role of ROS and subsequent oxidative stress as a pathological factor for IBD has not been well defined

This study identified ER as the major target for QIs-induced perturbations and a significant protective role of GSTP1 by alleviating loss of cell viability, enhancing detoxification of reactive QIs, and attenuating apoptosis