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Description

J Cell Physiol (2018) 233:642540

Intra-islet glucagon, acting through the beta cell GLP-1R and glucagon receptors, may also contribute to the incretin effect, perhaps reconciling findings of low circulating levels of GLP-1 yet substantial meal-stimulated GLP-1R-dependent potentiation of insulin secretion [45, 46]

Side Effects: Both are generally well tolerated, with minimal risk of side effects

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