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Although rodent models provide important tools for AD research, it is difficult for these models to fully simulate the complex pathophysiological processes of human AD because of the significant differences in brain structure, metabolic characteristics, and immune responses between humans and experimental animals (Qian et al., 2024)

Strong, unregulated growth signaling is useful in a petri dish and genuinely risky in a person which is the whole reason this belongs only in a medically supervised setting

( PMID: 15504997 ) Buse J, Montanya E, Sesti G et al (2010) The incidence of antibody formation and the levels of antibodies are lower with liraglutide than exenatide in a head-to-head comparison

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