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The oxidant/antioxidant imbalance is not peculiar to SARS, but it is shared by all inflammatory lung diseases in which the activation of redox-sensitive transcription factors such as NF-kB is reduced by GSH [37]

Combining mono- and/or dual-agonists to achieve triple agonism may allow for personalisation of treatment, aiming to find the optimal balance of receptor agonism for a specific individual

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