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Research confirms that -tocopherol synergizes with GPX4 through an electron donor-mediated chain-breaking mechanism to jointly maintain cellular membrane lipid redox homeostasis, thereby effectively inhibiting ferroptosis (146)

These primary antioxidants exist within our cells and are essential for cellular defence, detoxification and function

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In agreement, we observed that cellular GSH levels and the ratio of GSH/GSSG were increased in DMF treatment group (Fig