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Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling
9,394,338 Applications Treatment for Alzheimers and other neurodegenerative diseases Key Benefits & Differentiators Increased stability: Resistance to -glutamyl transpeptidase (-GT) breakdown leads to extended antioxidant activity Replicates endogenous GSH activity: Crosses the blood-brain barrier and mimics key glutathione (GSH) functions Technology Overview Oxidative stress and plaque formation from glycation are key features of numerous neurodegenerative diseases, including Alzheimers disease
