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To further understand the potential role of estrogen in regulating inflammatory stress, we stimulated two different types of macrophages (THP1-derived macrophages and liver resident primary Kupffer cells) with LPS in the presence and absence of estradiol and then analyzed the cytokine expression, IDO1, and Trp-catabolism

423 The research further demonstrated that this anti-inflammatory effect was not mediated by GLP-1R in the blood or endothelial cells but required the GLP-1R in the CNS