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After the Hp colonization, most mouse strains develop only mild inflammation but not GC

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pylori could colonize gastric epithelial cells due to its main components enabling it to survive in an acidic stomach environment and attach to host cells through the interaction of microbial adhesins with host cell receptors through flagellate-mediated motility

These actions resulted in a significant reduction in body weight, corresponding to liver and adipose tissue weight restrictions (73)