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Reducing SCFA-producing bacteria leads to intestinal anti-inflammatory and immunomodulatory dysfunction, and impaired glucose control

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Furthermore, renal tubule-specific deficiency of Acsl4 conferred significant protection against oxalate-induced AKI, as evidenced by alleviated structural and functional renal damage, reduced oxidative stress and decreased inflammatory cell infiltration, all of which collectively contribute to a reduction in ferroptosis

However, we still believe that this study reveals possible molecular mechanisms underlying UA mediated processes in macrophages that can be related to macrophage infiltration in adipose tissue in obesity