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Experimental models of anoxia/reoxygenation injury demonstrated that larazotide preserves epithelial barrier integrity by maintaining transepithelial electrical resistance and preventing the disassembly of tight junction components such as ZO-1, occludin, and claudins

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Consistent with the effects in rat PSCs, high glucose and TGF1 levels enhanced the proliferation and decreased the apoptosis of PSCs in the LG group, whereas GSH and SB431542 prevented the effects induced by high glucose