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While they share a common origin in growth hormone signaling, their mechanisms, specificity, and research applications differ significantly

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A Multi-Pathway Model We can think of this as a convergence of several mechanisms: L-cell activation from viral or microbial sensing Vagal signaling altered by cytokine release Gut-brain axis dysregulation, mirroring GLP-1 agonist effects Delayed motility from enteric nervous system inflammation Together, these pathways could produce a temporary GLP-1-like state during and shortly after the viral illness

[DOI] [PMC free article] [PubMed] [Google Scholar] Bothe J