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Postoperative glucocorticoid withdrawal syndrome is usually characterized by biochemical evidence of HPAA suppression, with many signs and symptoms consistent with cortisol deficiency despite the use of supraphysiologic doses of glucocorticoids

PINK1 KO in human and mouse DA neurons causes decreases in membrane potential and increases in ROS generation (Wood-Kaczmar et al., 2008)

[1] Patients should seek immediate medical attention for severe, persistent abdominal pain (which may radiate to the back), with or without vomiting, as these could be symptoms of acute pancreatitis

doi: 10.21203/rs.3.rs-1727226/v2 314 MartinLKoczeraPZechendorfESchuerholzT