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Renal fibrosis, the terminal pathological manifestation of most chronic kidney diseases (CKD), is a multifactorial process driven by extracellular matrix (ECM) dysregulation, fibroblast-to-myofibroblast transdifferentiation, immune cell infiltration, and tubular epithelial-mesenchymal transition (EMT)(Figure 1)

[DOI] [PubMed] [Google Scholar] 109.Shih C-C, Lee H-H, Chen T-L, Tsai C-C, Lane H-L, Chiu W-T, et al

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Moreover, GSH-independent roles of Gclc associated with its direct product-glutamylcysteine (-GluCys)might influence the downstream regulation of biosynthetic pathways of amino acids (and/or their transport) 119 in MZB and FoB