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A more general mechanism to explain oxidative stress generated by electrochemically silent metals is based on the formation of glutathione conjugates and on their decomposition under physiological conditions to yield two toxic agents: the nano-particulate metal sulphides that deposit in organ parenchyma and the electrophilic dehydroalanine analogue of glutathione

Transforming growth factor-1 inhibits interleukin-1-induced expression of inflammatory genes and cathepsin S activity in human vascular smooth muscle cells

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Some preclinical work explores FOXO4-p53-targeting peptides against senescent cancer cells, and the developer has discussed cancer as a research interest, but there is no human evidence that FOXO4-DRI treats cancer