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131 Because of its antioxidant properties, vitamin E had been proposed as a therapeutic agent, 139 , 140 , 141 but subsequent evaluation of large doses of the vitamin failed to demonstrate an ameliorative effect on anemia

Neuroprotection of catalpol for experimental acute focal ischemic stroke: preclinical evidence and possible mechanisms of antioxidation, anti-inflammation, and antiapoptosis

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Oxidative stress activates NUPR1 by both ER stress (METH [79] and H2O2 [80]) and non-ER stress pathways, NUPR1 regulates ferroptosis via iron metabolism, ROS homeostasis, and the GSH/GPX4 pathway [81, 82]