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Traditional risk factors such as dyslipidemia, hypertension, and smoking interact with SLE-related pathological mechanisms, including chronic inflammation, immune dysregulation ( Macrophages contribute to vascular injury through oxidized low-density lipoprotein (ox-LDL) uptake and foam cell formation ( + T helper 1 (Th1) cells migrate to arterial walls, promoting vascular inflammation, while B cells drive atherosclerosis via autoantibody production (e.g., anti-endothelial cell antibodies) and immune complex deposition ( Pro-inflammatory gut microbiota elevate systemic inflammation ( Emergencia timonensis accelerates atherosclerosis in mice and humans ( We have focused on discussing immune-targeted therapies (e.g., interferon inhibitors ( This review aims to offer novel insights and new research directions for atherosclerosis associated with SLE
Mishra AK, Parish CR, Wong ML, Licinio J, Blackburn AC
favorisce lincremento della capacit di contrazione e della gittata cardiaca , in parole povere il cuore riesce a pompare pi sangue con meno sforzo
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