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Key Benefits and Actions Potent methyl-free B12 and folate Methylation, energy, and cognition support Supports healthy homocysteine levels and

Under normal circumstances, mitochondrial glutathione rapidly neutralizes these byproducts

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Though pathogenic RFK mutations are rare, animal models have shown that RFK loss results in impaired mitochondrial electron transport, NF-B activation, cognitive decline, and neuroinflammation, highlighting its disease relevance