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For clinical strategies targeting ferroptosis, existing interventions include GPX4 inhibitors, such as red ginseng polysaccharides, which induce ferroptosis by downregulating GPX4

Therapeutics that block RSL3-mediated GPX4 inactivation may provide a targeted approach to preserving antioxidant capacity in ARDS patients, reducing the extent of ferroptosis-driven lung damage ( 5.3 Iron chelators Iron overload exacerbates ferroptosis by fueling ROS production and lipid peroxidation, making iron chelation a viable therapeutic approach

The honest limits on both: Neither molecule has run a human lifespan trial

In mice subjected to a methionine-choline-deficient diet, vitamin E antioxidant therapy restored GSH levels (Phung et al