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CONCLUSION This review aimed to elucidate the principal mechanisms involved in the neuropsychiatric manifestations of COVID-19, which involve glutamate dysregulation, Xct, Nf-B, mGluR2, and mGluR3, astrocyte and glial damage and neurotoxicity highlighted as central factors [15], exploring the potential of NAC and ALC as promising therapeutic agents in neuropsychiatric conditions COVID-19-related, including those associated with PCS
However, due to the bidirectional regulation mechanism of ferroptosis, targeted therapy for ferroptosis is still in its infancy
This is significantly longer than peptides reconstituted in plain sterile water, which typically degrades faster without the bacteriostatic preservative
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