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PC molecules in HDL interact with ATP-binding cassette transporters, facilitating the efflux of cholesterol from macrophages in arterial walls to HDL particles

3,4 In contrast, in Parkinson's disease (PD), neuronal deposits of -synuclein are present and form the primary structural component of Lewy body fibrils that are characteristic of the disorder, whereas in Huntington's disease (HD), cytoplasmic and nuclear deposition of the huntingtin protein and fragments thereof are found