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Proposed oncogenic mechanisms involve chronic inflammation, direct virulence factor activity (CagA/VacA), and microbiome dysbiosis

reported that the low GSH production of permeated cells immobilized using a polyacrylamide gel was caused by the low efficiency of the ATP supply: only 0.5 % of the total regenerated ATP from the glycolytic pathway was actually used for GSH synthesis in E

They also found that ROS damage affected the nucleus of the sperm causing DNA damage which could be transmitted to the next generation and be responsible for increased disease susceptibility in offspring

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