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Still, conventional or alternative, there is no magic bullet when it comes to long COVID

These findings demonstrate that VPA exposure induces significant microglial activation in the hippocampal CA1 region, which is effectively attenuated by hCiPSC-NSCs treatment, indicating a substantial reduction in neuroinflammatory responses

When autophagy is impaired, increased p62 competes with the DLG motif for binding to the Keap1 DC pocket and inhibits the ubiquitination of Nrf2, causing Nrf2 stabilization and the increased expression of cytoprotective genes