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It has been demonstrated that in animals with ALDH4A1 mutation, vitamin E and vitamin C supplementation can reverse the inhibition of ATPase activity by hyperprolinemia, and appropriate use of antioxidants can also be helpful to reduce neurotoxicity [19]

This GHR upregulation sensitises local cells to circulating GH, potentiating GH-induced JAK2/STAT signalling and stimulating collagen matrix synthesis, IGF-1 production, and fibroblast proliferation providing a mechanism through which BPC-157 engages the GH/IGF-1 axis at the tissue level without altering systemic GH secretion

& Chang, J