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Our previous study suggests the predominant involvement of quinone products in long-term DA-mediated inactivation of complex I and IV(1)

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J.de BoerR

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Nicholson and Kullmann, 2014)

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In summary, CRAT functions as a critical tumor suppressor in OC progression by enhancing PGC-1-mediated mitochondrial biogenesis and metabolism, suggesting CRAT as a potential therapeutic target in treatment of OC

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