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These effects arise from both direct microbehost interactions. Dysbiosis markedly diminishes the physiological tolerance of intestinal tissues to ferroptosis, thereby compromising the intestinal barrier function and exacerbating colitis (114)

Thus, epithelial cellexpressed TMPRSS2, which critically contributes to the lung pathology in SARS-CoV-2 infection, emerges as an intestinal incretin regulator and a potential link between infection and chronic cardiometabolic diseases

Olorofim for the treatment of invasive fungal diseases in patients with few or no therapeutic options: a single-arm, open-label, phase 2b study

DSIP Peptide Key Points The central distinctions needed to understand DSIP research responsibly