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In this view, increased oxidative stress has been found to enhance hypercholesterolemia, hypertension, diabetes mellitus, and chronic renal failure [6, 7]

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139 Initially, it was hypothesized that the loss or reduction of ABCG2-mediated renal urate secretion would result in increased renal urate reabsorption, as diminished renal excretion is typically considered the primary mechanism of hyperuricemia in most gout patients

Note, exposure in mg/kg should be based on Ideal Body Weight