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gondii infection activates ferroptosis in mice, causing pathological damage ( Table 3 summarizes the roles of ferroptosis in fungal and parasitic infections, highlighting distinct regulatory mechanisms across different pathogens

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Upon stimulation by agents such as VEGF, the binding of eNOS and Cav-1 is released, eNOS associates with calmodulin, and then subsequently phosphorylated by heat shock protein 90 and AKT 23