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Tirzepatide activates both GLP-1 and GIP pathways, while semaglutide primarily activates the GLP-1 pathway

Studies utilizing Alzheimers disease models suggest that semaglutide treatment inhibits the activation of microglia and astrocytes, thereby reducing the release of pro-inflammatory mediators ( 5.2 Enhanced brain metabolism and insulin sensitivity The association between Alzheimers disease (AD) and metabolic dysfunction has led to the characterization of AD as Type 3 diabetes, a condition marked by impaired insulin signaling and glucose utilization in the brain ( 5.3 Direct neuroprotective and pro-survival pathways In addition to its anti-inflammatory and metabolic effects, semaglutide activates pathways that directly enhance neuronal survival and resilience

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The IGA and Investigator Overall Improvement (IOI) evaluations were performed on a 5-point scale and transformed to numerical values (05) and the mean, standard deviation and percentages were calculated