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Targeting these amino acid-driven metabolic circuits may offer novel therapeutic opportunities in fibrotic diseases such as IPF, where metabolic vulnerabilities can be exploited to disrupt fibroblast activation and excessive ECM remodeling
A key process in glomerulosclerosis and tubular epithelial fibrosis involves the epithelial-to-mesenchymal transition (EMT) induced by high glucose levels (Xu et al., 2017)
We all have unique environmental/lifestyle factors and specific dietary or genetic considerations
doi:10.1007/s10815-012-9714-1
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