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Considering the central role of glucose catabolism for stimulus-secretion coupling in -cells, our findings suggest that chronic GLP-1 actions on insulin secretion include elevated -cell glucose metabolism

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doi: 10.1074/jbc.M610737200

Dysfunction of mitochondria and increased levels of oxidative stress in the brain, neuronal cell cultures, urine, and erythrocytes have been confirmed in individuals with Down syndrome (Jovanovic et al