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GLP-1 receptor agonists are cleared via renal pathways, and their use in patients with moderate-to-severe chronic kidney disease (CKD) requires careful consideration

van Buul-Offers et al (1995) have shown that overexpression of IGF-2 in transgenic normal and pituitary deficient Snell dwarf mice causes increased growth of the thymus, suggesting a role for IGF-2 in thymic development by paracrine or autocrine mechanisms